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Wohlfarth-Gruppe geht auf die BruderGruppe über . Placental explants were exposed to adiponectin (250 ng/ml) or FCS (10%) for 48 h. Following incubation, explants were fixed, paraffin embedded, and stained for anti-MKI67 antigen, reflecting proliferation of cells as described in Effects of adiponectin on cell proliferation in human first-trimester placental explants. Blastocyst-endometrium interaction: intertwining a cytokine networkHormonal and paracrine regulation of embryonic implantation: a workshop reportThe ‘beneficial' adipokines in reproduction and fertilityHuman chorionic gonadotropin stimulates trophoblast invasion through extracellularly regulated kinase and AKT signalingDevelopmental changes in mouse placental cells from several stages of pregnancy in vivo and in vitroInsulin-like growth factor-I and -II regulate the life cycle of trophoblast in the developing human placentaLeptin expression in placental and fetal tissues: does leptin have a functional role?The effect of leptin on mouse trophoblast cell invasionTransduction of leptin growth signals in placental cells is independent of JAK-STAT activationLeptin promotes cell proliferation and survival of trophoblastic cellsLeptin modulates extracellular matrix molecules and metalloproteinases: possible implications for trophoblast invasionDual roles of adiponectin/Acrp30 in vivo as an anti-diabetic and anti-atherogenic adipokineAdipocytokines: mediators linking adipose tissue, inflammation and immunityAdiponectin promotes adipocyte differentiation, insulin sensitivity, and lipid accumulationSelective purification and characterization of adiponectin multimer species from human plasmaT-cadherin is a receptor for hexameric and high-molecular-weight forms of Acrp30/adiponectinCloning of adiponectin receptors that mediate antidiabetic metabolic effectsAdiponectin stimulates human osteoblasts proliferation and differentiation via the MAPK signaling pathwayAdiponectin activates c-Jun NH2-terminal kinase and inhibits signal transducer and activator of transcription 3Secretion of adiponectin by human placenta: differential modulation of adiponectin and its receptors by cytokinesExpression and regulation of adiponectin and receptor in human and rat placentaExpression of adiponectin receptors and its possible implication in the human endometriumPermissive action of glucocorticoids on catecholamine-induced lipolysis: direct “in vitro” effects on the fat cell beta-adrenoreceptor-coupled-adenylate cyclase systemDifferential effects of inducers of syncytialization and apoptosis on BeWo and JEG-3 choriocarcinoma cellsAdiponectin mediates an antiproliferative response in human MDA-MB 231 breast cancer cellsA rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye bindingRapid nongenomic E2 effects on p42/p44 MAPK, activator protein-1, and cAMP response element binding protein in rat white adipocytesAdiponectin mediates antiproliferative and apoptotic responses in human MCF7 breast cancer cellsActivation of AMP-activated protein kinase increases mitochondrial enzymes in skeletal muscleProadipogenic effect of leptin on rat preadipocytes in vitro: activation of MAPK and STAT3 signaling pathwaysAdiponectin receptors in human adipose tissue: effects of obesity, weight loss, and fat depotsSomatostatin as a regulator of first-trimester human trophoblast functionsAdiponectin stimulates angiogenesis by promoting cross-talk between AMP-activated protein kinase and Akt signaling in endothelial cellsAdipocyte-derived plasma protein adiponectin acts as a platelet-derived growth factor-BB-binding protein and regulates growth factor-induced common postreceptor signal in vascular smooth muscle cellEffects of adiponectin on breast cancer cell growth and signalingPost-translational modifications of the four conserved lysine residues within the collagenous domain of adiponectin are required for the formation of its high molecular weight oligomeric complexRegulation of adiponectin and its receptors in rat ovary by human chorionic gonadotrophin treatment and potential involvement of adiponectin in granulosa cell steroidogenesisThe regulation of adiponectin receptors in human prostate cancer cell linesAdiponectin and its receptors are expressed in adult ventricular cardiomyocytes and upregulated by activation of peroxisome proliferator-activated receptor gammaAdiponectin expression in human fetal tissues during mid- and late gestationCord plasma adiponectin: a 20-fold rise between 24 weeks gestation and termLeptin secretion to both the maternal and fetal circulation in the ex vivo perfused human term placentaAdiponectin-induced antiangiogenesis and antitumor activity involve caspase-mediated endothelial cell apoptosisAdiponectin and its receptors are expressed in bone-forming cellsAdiponectin as a growth inhibitor in prostate cancer cellsSelective suppression of endothelial cell apoptosis by the high molecular weight form of adiponectinSyncytial fusion of human trophoblast depends on caspase 8Leptin and adiponectin interact in the regulation of prostate cancer cell growth via modulation of p53 and bcl-2 expressionAdiponectin blocks multiple signaling cascades associated with leptin-induced cell proliferation in Apc Min/+ colon epithelial cellsSerum adiponectin and leptin levels in Taiwanese breast cancer patientsPlasma adiponectin concentrations in non-pregnant, normal and overweight pregnant womenCirculating levels of adiponectin and leptin at 23–25 weeks of pregnancy in women with impaired placentation and in those with established fetal growth restriction© 2009 by the Society for the Study of Reproduction, Inc.

Addamsarjan is one of the millions playing, creating and exploring the endless possibilities of Roblox. 1B).Because phosphorylation-dependent activation of PRKA is the major transduction pathway reported for adiponectin signaling, the phosphorylation of PRKA was investigated in JEG-3 and BeWo cellular extracts. Intra-abdominal adipose tissues were obtained from patients undergoing elective abdominal surgery (mean ± SEM age, 59.5 ± 4.3 yr). Search for other works by this author on:

The direct effects of adiponectin on growth capacities of JEG-3 and BeWo choriocarcinoma cell lines, commonly used as models for first-trimester trophoblast cells, were investigated.Dulbecco modified Eagle medium (DMEM), DMEM/F12, penicillin, streptomycin, HEPES, leupeptin, aprotinin, 5-aminoimidazole-4-carboxamide 1β-D ribofuranosyl 5′monophophate (AICAR), 4-(2-aminoethyl)-benzene-sulfonyl fluoride (AEBSF), NaF, and bovine serum albumin (BSA) were purchased from Sigma Chemical Co. (St. Louis, MO). Mitogen-extracellular kinase (MEK) inhibitor UO126 was from Promega (Madison, WI), and PIK3 (PI3K) inhibitor LY294002 was from Calbiochem (Merck Chemicals, Nottingham, England). Pachymic acid protects oocyte by improving the ovarian microenvironment in PCOS mice Effects of adiponectin on PRKA activation in BeWo (For the following studies, the intermediate concentration (250 ng/ml) of adiponectin was used. UO126 and LY 294002, two specific inhibitors of these pathways, suppressed the effects of adiponectin on [In conclusion, we show herein that adiponectin may act as an antiproliferative factor in human trophoblastic cells and provide examples of cells affected by the opposite actions of leptin and adiponectin. Second, we demonstrated that adiponectin induces a reduction in cell number and in [Embryo implantation and placentation are two critical steps for establishing pregnancy. Statistical analysis was performed using the nonparametric paired Wilcoxon test or unpaired Mann-Whitney We aimed to investigate the effects of adiponectin on BeWo and JEG-3 cells. Cells were exposed for 24 h to adiponectin (250 ng/ml), the MEK inhibitor UO126 (10 μM) with or without adiponectin (250 ng/ml), or the PIK3 inhibitor LY294002 (10 μM) with or without adiponectin (250 ng/ml) in the presence of [First, we investigated whether BeWo and JEG-3 cells were target cells for adiponectin and demonstrated (for the first time to our knowledge) that ADIPOR1 and ADIPOR2 (but not T-cadherin) were expressed in JEG-3 and BeWo cells. Removal of peri-ovarian adipose tissue affects follicular development and lipid metabolism

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